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A later ape probably ancestral to gibbons. Dryopithecus (mid-Miocene) -- A later ape probably ancestral to the great apes & humans. At this point Africa & Asia connected via Arabia, and the non-gibbon apes divided into two lines: |
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At last, a good mutation? There are children who are so prone to infection that, if they survive at all, they have to spend their lives in an artificial ‘bubble’. This is the usual fate of those who have inherited two defective copies (one from each parent) of a gene which produces an enzyme called ADA (adenosine deaminase). Because they are unable to make ADA, toxic substances accumulate in their blood which slowly damage the body’s immune cells. However, in an unprecedented finding, a U.S. boy called Jordan Houghton has spontaneously recovered from his condition.1 All the evidence indicates that in one line of his immune cells, one of the faulty genes has apparently repaired itself. Geneticist Hagop Youssoufian at Brigham and Women’s Hospital, Boston, says about this 'fascinating' occurrence: ‘We finally have a clear example of a mutation doing something good’. ‘Back mutations’, replacing a letter in the DNA sequence which was faulty back to what it originally should have been, are not unknown. They certainly do not show us how significant information can arise de novo, as they merely (accidentally) ‘restore’ what should have been there.An occurrence like this (encouraging, but exquisitely rare) may actually not be mutational as such, as there are abundant error-checking, proof-reading and repair mechanisms in our genetic machinery. Youssoufian’s ‘at last’ statement highlights the fact that mutations, random accidental changes in copying hereditary information, are overwhelmingly a downhill process. Geneticists in hospitals are all too familiar with the harm they cause in people who inherit their effects. |
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ARE MUTATIONS HELPFUL? Because natural selection can only choose from what is there, evolutionists must have faith that somehow mutations provide the new raw material for living things to evolve into other, more complicated life-forms having new structures and functions. But this is a blind faith, completely unsupported by the facts. The blueprint of living things earned by DNA is more complicated than the most sophisticated computer program. This blueprint is copied when living things reproduce, and mutations are nothing more or less than chance mistakes during copying. Can you imagine trying to improve a computer program, to give it new, more complex functions, by relying on copying mistakes? That's why the thousands of mutations of which we know in the human race are labelled by the diseases they cause. Listen to what the evolutionist Dr Pierre-Paul Grasse, Europe's most distinguished biologist, has to say. He was past president of the French Academy of Sciences, and editor of the prestigious 26-volume Traite de Zoologie'. 'The opportune appearance of mutations permitting animals and plants to meet their needs seems hard to believe. Yet the Darwinian theory is even more demanding: A single plant, a single animal would require thousands and thousands of lucky, appropriate events. Thus, miracles would become the rule: events with an infinitesimal probability could not fail to occur.' -- Pierre-Paul Grass' |
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Evolution, Creation, and Thermodynamics Introduction: The Second Law of Thermodynamics states simply that an isolated system will become more disordered with time In the first part of this article, we established that the naturalistic self-transformation of the universe from simple to complex required by evolution is in direct contradiction to the second law. Known "rules" of thermodynamics render the evolutionary origin of stars and planets from condensing clouds of gas implausible. Many evolutionists claim however, that the earth is an open system and local increases in order are possible, eg the observation that ordered crystals form spontaneously from less ordered solutions means that evolution from simple to complex can occur. This article will investigate these claims. We have seen that in a number of open systems, order would apparently increase by Itself. Let's take three examples: 1. A seed growing into a plant 2. Workmen building a car 3. Saltwater cooling down to form salt crystals The Second Law is not violated in any of these, since the total disorder in the universe increases as follows: CHANGE IN OPEN SYSTEM II Disorder Decreases + CHANGE IN SURROUNDINGS II Disorder Increases EQUALS CHANGE IN WHOLE UNIVERSE ("Isolated system") II Disorder Increases The decrease in disorder of the open system's more than balanced by the increase in disorder in the surroundings, so that the disorder in the whole universe always increases, e.g. when the saltwater cools, it heats up the air around it, which gives an increase in disorder in the air molecules. Therefore, says the evolutionist, you can have a local decrease in disorder (eg., on the earth) balanced by an increase in disorder elsewhere, without violating the Second Law. So far, he is right, (if we ignore the fact that the chaos to cosmos notion is invalid when we consider the whole universe) except that even a local increase in order will not happen unless we have special conditions. Order, complexity and information will never arise spontaneously without a mechanism or motor. Take Example No. 1. The raw energy pouring from the sun onto the seed will produce disorder, not order, unless the seed has the highly complex photosynthetic mechanism and the direction of the genetic code. A seed growing into a plant is not analogous to the presumed evolutionary process in any case, since it involves an 'unfolding' of information which is already there in the genetic 'blueprint'. Evolution requires information and complexity to arise and keep increasing over millions of years. In Example No. 2 we also have an open system and available energy, but again we have an energy conversion mechanism, and coded information giving direction to the process. We see that it takes machines to make machines — it takes ordered systems to produce ordered systems. In living things, the information necessary to overcome the effects of the Second Law is passed on from generation to generation. This information 'rides on' the chemistry of the cell, just as the information in this article 'rides on' the ink and paper, but transcends it The information in the DNA code and this page both depend on the sequence, or specific order of the constituents. The Second Law tells us that this can be copied many times, eg., in a photocopier, but information will never spontaneously be added to it — rather it will tend to be lost. The original information on this page had to be imposed upon it from the outside and had its origin in MIND — just as the information in the genes of living creatures had its ultimate origin in the mind of God and was imposed upon the matter in Creation Week. On the "primitive earth" there could have been no machines or ordered systems — the first "primitive cell" could not arise without these special conditions, as we have seen. Example No. 3 (crystals) is often cited, but has no relevance to the problem. This is because biological growth processes involve complexity, whereas crystal growth involves regularity If you break up a large salt crystal, you get a lot of smaller salt crystals. If you break up a molecule of a biological protein, eg. insulin, into smaller pieces, it is no longer insulin since the information it carries in its specific sequence of components is lost. A crystal of ice, for example, carries no more information than a single water molecule. The formation of a crystal involves molecules assuming a rigidly predetermined pattern — there is no growth in information or complexity, and again there is a pre-existing "code". For the sake of further discussion, let's allow the first cell to somehow form in violation of these facts. Obviously, until you have something living and reproducing, mutation and selection are not involved. Could mutation and selection act as the necessary mechanism/code to locally overcome the effects of the Second Law? Mutation is a random change in a pre-existing code It is not, therefore, a code or a mechanism as such. Selection is merely a commonsense occurrence — the elimination of the unfit. It cannot be either a code or an ordering mechanism in itself. What about both together? The evolutionist still has one counter-argument left, providing we ignore the impossibility of getting to the primitive earth and the first cell. The minute random fluctuations in order represented by genetic mutation are "fixed" and given a certain direction by natural selection, he claims Thus, the two acting in concert act as a mechanism; the analogy is occasionally given of a jack, where the handle moves up and down, and natural selection is represented by the ratchet, "lockingin" those motions which are in the right direction. Dr. Harold Armstrong, a physicist and editor of the Creation Research Society Quarterly, correctly points out that this superficially attractive analogy is not appropriate, since the handle movements are not truly random, but directional — ie., up and down. A closer analogy, he claims, would be as follows: The random motion of electrons in the resistor A at a particular room temperature would cause some to flow in the direction of the arrow. The rectifier B would only allow those in one direction to pass, and thus a current could flow, driving electric motor C which could perform useful work. It sounds good, but it won't work. This machine would be continually extracting heat from the environment to perform work, and one of the consequences of the Second Law is that this can't happen. This example deserves further consideration by creationists — a detailed analysis, considering e.g. fluctuations in order in the rectifier itself and applying these to the bioloqical situation may be fruitful. A further point is that this classic "small fluctuations" argument of micromutation is in serious trouble on other grounds (the absence of transitional forms, the difficulty accounting for the "usefulness" of proposed transitional stages, and the small amount of genetic "load" in living things) which are forcing a number of leaders in evolutionary thought back to "macromutations" (sudden leaps or "saltations" — e.g. a non-flying creature becomes a flying one in one single mutation). Yet to get out of one set of difficulties, they must propose that a random change has given rise to a significant increase in order and information — the Second Law says that this will not happen without a mechanism which in this case is certainly lacking. In conclusion, 1. The Second Law applied to the whole universe is the death-knell for any proposed evolutionary scheme. (see part 1) 2. No biological order can arise without pre-existing coded mechanisms — the formation of the first cell from naturalistic processes is a thermodynamic impossibility. 3. After the first cell, mutation/selection do not appear to be adequate candidates for the ordered mechanism required to locally overcome the effects of the Second Law in an open system. Information and order, form, body, arrangement and complexity do not arise spontaneously, but are spontaneously and naturally lost. |
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Can you imagine trying to improve a computer program, to give it new, more complex functions, by relying on copying mistakes? |
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Probably... un termen foarte indragit de evolutionisti. |
QUOTE ("Catalin") |
repetind probcesul acesta de multe ori si folosind destui "indivizi" se pot "evolua" programe. |
QUOTE ("Catalin") |
Evident, in urma "mutatiilor" apar multe programe care au erori de compilare. |
QUOTE ("Catalin") |
Exista indivizi cu erori, nu specii intregi. |
QUOTE ("Mi_") |
A, si pentru supravietuirea speciei în sine, mutatiile sunt amenintari, nu conditie ca sa supravietuiasca. |
QUOTE ("Mi_") |
Si nu-i nevoie sa-ti explic asta, e suficient ca mutatii aleatoare au loc zilnic, fiecare având o sansa (mica, mare, simpla, complexa) sa determine o schimbare. |
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Webster defines dilemma as a problem "seemingly incapable of a satisfactory solution." The problem for evolutionists is to develop a theory of origins that would account for the intricate design mechanism of biological pollination. Evolutionists believe that in order to attract pollinators, some plants have adapted to bribe insects, birds and other animals with a meal of nectar or pollen. The basic tenet of evolutionary theory is there is no intelligence involved in the process. One cannot have it both ways. Either an intelligent personal Designer planned and executed the process of biological pollination or it just happened to occur in some mindless, wasteful chaotic pipe dream. Let's take a logical look at the possibility of an evolutionary development of biological pollination. In order for the process to work, one must start with a plant that needs pollinating. There is a mutual benefit between plant and pollinator who needs the nutrients the plant offers. If plants were able to reproduce before the pollination process evolved, would a world of survival of the fittest be the environment suited for such a process? If a random chance mutation produced a plant that needed pollinating, would that mutation be the most apt to survive? Absolutely not. The plant would have to share soil, rainfall and nutrients with other plants that were able to reproduce without pollination. These plants would complete their life cycles, bear fruit and crowd out the mutant before the first pollen grain located another plant that had evolved the egg that awaited fertilization. Design of the Pollen Grain Pollen differs from plant to plant. Its macroscopic millions are carried by wind, insects, birds, and other animals. Evolutionists claim that fossilized remains of pollen prove it has remained much the same for 120 million years. In other words, pollen is never used as evidence that long periods of time produce evolutionary changes. When confronted with evolution's violation of the true scientific method that demands observation or repeatability, evolutionists are quick to raise the banner of endless eons of time. But when evolutionists dig a moat of the endless ages between logic and the theory it threatens, remember that pollen has endured unchanged throughout the ages of this imaginary history. Sunflower pollen looks like a spiked ball, while the pollen of the black salsify looks somewhat like a diving bell with pentagon shaped windows. (The roots of some salsify plants are edible.) Dandelion pollen looks more like a knight's weapon than the casing of DNA that grows a child's plaything in a spring meadow. Pollen is so distinctive, it has been used to confirm a murderer was at the scene of a crime; the irrefutable evidence led to his confession. Growth of the Pollen Tube The fertilization of a flower does not take place on the tip of the stigma (see diagram). The life-giving egg is hidden well inside the style of a plant. Evolutionists have made the claim that plants evolved this placement of the egg in order to keep insects from eating the viable part of the plant. The placement of the egg is significant when one considers what the pollen must do to reach it and fertilize the plant. The pollen has the physiological information resident within it to build a tunnel to the egg. It is called a pollen tube. It is macroscopic in size. Pollen tubes grow at different rates, but some have been reported to grow as much as 1.5 inches per hour. A mature grain of pollen consists of three cells, two sperm cells and one vegetative cell. A generative cell forms the sperm cells. In most plants the generative cell completes its division while in the pollen tube. Under a microscope, one can see the cilia of these cells in the cycad known as Zamia bear the unmistakable logarithmic spiral of a chambered nautilus. Self-Incompatibility In order for a seed to grow, it must be fertilized by pollen from another bloom. Scientists readily admit they do not understand how this works altogether, although they have learned there are incompatibility proteins in both the pollen and the stigmas or styles. When proteins from the pollen interact with proteins from the same bloom's stigma or style, fertilization is prevented. Remember, evolutionists say that pollen has remained unchanged for 120 million years. If that is the case, why haven't the plants adapted to stop producing pollen that must be carried to another plant? Why couldn't the plant "learn" to accept its own pollen? That would certainly be at least as easy as learning what kind of nectar or pollen would attract the correct kind of carrier to insure a load of pollen reached a neighboring plant! Preventing Self Pollination Plants have a variety of mechanisms to prevent self fertilization. The wild geranium ripens all of its stamen first. When the pollen from the stamen is all gone, the stigma ripen. In a monoecious plant, one plant (mono) produces both types of blossoms. Some blossoms will have only the stamen (with pollen) and others will have only stigma (with the eggs). The blooms may looks very similar (as in the case of a squash plant) or very dissimilar, as in the case of the hazel tree. On the hazel tree, the stamen is a long catkin (flexible thin tassels several inches long). The stigma is a tiny pink flower-shaped spray on the same tree. The hazel tree is pollinated by the wind. As the catkins wave in the breeze, they scatter the pollen and it sticks on the tiny pink sprays. A dioecious plant has all stamens on one individidual plant and all stigmas on another individual plant (cottonwood, green ash and the box elder trees). What if the plant self pollinates? In experiments with foxtail grass, it was shown that once the pollen was recognized as incompatible, it was rejected and the pollen tube filled again with plant tissue. Incredible as it seems . . . The Indian Balsam plant is an example of the incredible relationship between the pollanator and the plant. An insect enters the balsam to get a drink of nectar. It brushes pollen from stamen that is attached to the bottom of the stigma. The insect that carries away the final load of pollen also carries the stamen away and exposes the stigma, which is then ready to be pollinated by the next visitor. The Dutchman's pipe lures gnats into its large cavernous curving structure with a scent similar to a fungus on which they feed. Once a gnat enters one end of the curved structure, it mistakes the dead-end bottom for the exit because of its brightness. If the unwary gnat is carrying a supply of pollen, the fertilization of the Dutchman's pipe also triggers a mechanism that lowers its vase-like curve, opens it to the light and allows an easy exit for the gnat. In the orchid, the stamen is fused with the style and stigma. The pollinium is a structure of the orchid that combines the anther and the filament into one. The orchid is designed so that one of its petals is a "landing platform." The insect pollinator is compelled to land in a particular spot and and the trip to the nectar orchestrates his pick up of the pollinium. The pollinium accompanies the insect traveler on its next visit to a neighboring orchid where, of course, the strategic location of the landing platform will cause the pollen-laden pollinium to brush the surface of the stigma. If the pollinium of the second orchid has not yet been pulled away by an insect, the stigma is not yet exposed, so pollination will not take place. The color of a flower can give clues to what carrier is involved in the pollination of its plant. Most plants with flowers in the red spectrum are bird pollinated. Insects, who far outnumber birds as pollinators, are more attracted to the blue spectrum. A bee's eyes are sensitive to the ultraviolet spectrum. In fact, the yellow of a marsh marigold is seen as a beautiful iridescent color known as "bee purple." The structure of many flowers is exactly suited to the insect that pollinates them. The butterfly is equipped with a long roll-up tongue that is especially suited to the thin throat of many flowers (the bougainvillea, for example). The gentian plant of South Africa can only be pollinated by a Carpenter Bee which has a particular resonance it uses to vibrate the pollen from the flower it visits. Bees that do not match this resonance cannot pollinate the flower. One would think with the vibration of the pollen, it would be scattered everywhere and self-pollination would be a problem. Not so. The stigma is not receptive until all the pollen is gone. In Conclusion Any study of life yields evidence of the Creator's handiwork. Botany is a feast in this regard. Any parent or teacher who gives time to its study will enrich the lives of the children within their sphere of influence. Charlotte Mason said it best: "Any woman who is likely to spend an hour or two in the society of children, should make herself mistress of this sort of information; the children will adore her for knowing what they want to know, and who knows but she may give its bent for life to some young mind destined to do great things for the world." |
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Am inteles de la bun inceput explicatia logica a notiuni de camuflaj, insa asta nu inseamna obligatoriu evolutionism. |
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Daca natura incearca trei variante de blanuri pentru ursi atunci unde gasim ursii albastrii sau verzi intermediari, prin care aveam dovada ca s-a incercat de la bun inceput cate ceva? |
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Intrebarea mea era alta. Prin ce mister "vede" blana ursului culoarea alba a zapezii? |
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Problema evolutionismului e timpul. |
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Teoretic poti spune ca ursul brun a devenit alb in milioane de ani, insa intrebam daca timpul de milioane de ani permite si supravietuirea speciei pana cand Mama Natura isi termina incercarie de adaptare. Cum a vanat ursul brun sau verde in imensitatea alba pana a "imprumutat" culoarea zapezii? Cum a reusit sa supravietuiasca? |
QUOTE ("Mi_") |
Nu blana ursului vede culoarea zapezii ci focile vad mai clar alta culoare decât a zapezii, pe fondul alb al zapezii, si devin prada mai dificila. |
QUOTE ("Mi_") |
a aparut prin mutatie un urs cu blana alba, care spre completa lui mirare a vazut ca daca se duce pe banchiza poate prinde si foci. |
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The Dynamic Genome The term "evolution" is an oxymoron. The reality of genetic adaptability, doesn't equate with Darwin's fantasy. Every prototype genome possesses inherent genetic adaptability that assures a kaleidoscope of descendant diversity. Some public minds mistakenly confuse the reality of nature's genetic dynamism with evolutionism's make-believe. Human DNA carries 3+ billion base pairs. Two human parents possess the capacity to produce 70 trillion variations in their children. Darwin's Galapagos finches produced offspring endowed with different shaped beaks---but 21st century Galapagos finches still fly as finches! Fruit flies, subjected to laboratory induced mutations, may add or subtract wings and legs but continue producing fruit flies, ad infinitum---never butterflies nor dragonflies! Extrapolation doesn't mend the "flaws" and "holes" of Darwin's "rag of an hypothesis." Big Bang! The origin of matter by an explosion in empty nothingness an alleged fifteen billion years ago baffles. Something from nothing? Billions of cosmic bodies carving predictable orbits originating in an explosion? Since when does a horrendous explosion create order---floating in space? Darwin avoided attempting to explain the origin of inorganic matter or the process of the first appearance of organic life from non-life. Spontaneous Generation Nineteenth century, primitive wisdom mistakenly viewed single-celled, organic life forms as "simple." A blob of protoplasm! In reality, a living cell is vastly more complex than the most sophisticated man-designed mechanism. No human laboratory has duplicated what random chance allegedly accomplished in some still unidentified, "prebiotic soup." No evidence exists that original life defied impossible odds to create itself by spontaneous generation. NEVER!!! Random Chance Odds The odds of random chance generating an ecologically unique environment essential to produce and sustain organic life on Planet Earth is mathematically less likely than 6-billion blindfolded humans simultaneously solving a Rubik cube puzzle---three times in a row. Amino-Acid Building Blocks are Not Ambidextrous A cell's proteins require "left-handed," amino-acid building blocks. DNA & RNA use only "right-handed" building blocks. Natural forms come in a 50-50 mixture. Evolutionism's random chance explains neither the cause nor the discriminating process that mandates selection of left-handed v. right-handed building blocks. Information The specific kind of life reproduced by each genome is shaped by the information packed into the genes of each microscopic cell. Evolutionism does not account for the original source of that information vested in the dynamic genome of evolutionism's first ever cell from its beginning.. Mutations Mutations, genetic mistakes, the alleged engines driving evolutionism, are rarely beneficial. Mutations usually degrade the genome and trigger information loss. These genetic defects do not introduce new information to the gene code. Nor do genetic defects represent an incremental step in a mythical leap to some radical new organism. Variety potential, inherent within the dynamic genome assures genetic adaptability but not some quantum leap to some radically new prototype life. Neo-Darwinism ignores this scientific reality and relies on billions of mutations, combined with natural selection, as the source of every kind of life on earth after that first life cell. Natural Selection Counting on natural selection to combine with mutations to produce the engine of evolutionary change, creates a major league dilemma: the exact opposite results. Thanks to natural selection, mutations tend to be weeded out rather than serve as a conduit for the development of an entirely new life-systems!!! Inflated Time Without mega-chunks of time, evolutionism bites the dust as just another fictional whim. Radiometric dating's anomalous results raise red flags. Washington State's Mount St. Helens blew its top in 1980. Radiometric dating of the event produced conflicted results ranging from 340,000 years before the present to as long ago as 2,800,000 years BP. Assertions as to the age of the earth deserve caveats. Without those mega-chunks of time, mega-evolution qualifies as so much wishful thinking. But with those chunks of time, where are the billions and billions of people that by now would be walking on each other's toes? Two thousand years ago, an estimated 100 million Homo sapiens lived on Planet Earth. Thanks to geometric progression, the numbers have spiked radically with an estimated 6 billion of the human family on hand who welcomed the 21st century! If the earth is even 100,000 years old, where are all the people that would be bursting the seams of the planet if the past can be measured by the present? Abrupt Appearance Contrary to the gradual, incremental "progress toward perfection" conjectured by Darwin's mega-evolution, thousands of fully formed, intelligently designed and irreducibly complex life forms appeared abruptly, simultaneously and world-wide, without persuasive fossil evidence of prior ancestry. This Cambrian Explosion defies the anchor premise of Darwinian conjecture. Stasis Following this across-the-board sudden appearance of thousands of distinctly different kinds of life, descendants of these unique life forms continue to inhabit planet earth, virtually unchanged. Undeterred by mega chunks of deep time conjectured by evolutionism, bacteria continue to generate bacteria, fish produce fish, and apes parent apes. Coelacanths still swim in marine waters and Wollemi pines still grow on land. Missing Transitionals Evolutionism requires billions of incremental steps to bridge the gaping genetic chasms between organic prototypes: single cell-to-fish-to- amphibian-to-reptile-to-bird and mammal and eventually, to-man. After billions of fossil discoveries, where is the evidence of Darwin's "…innumerable transitional forms…"? Certainly not in today's living natural world. As to the miniscule number of fossil forms categorized as "transitional," they represent just another extinct species. So-called "intermediates" carry the label because an evolutionist has declared it, not because of irrefutable proof of genetic linkage as between old bones. Neither homology nor morphology equates genealogy. Irreducible Complexity A partially evolved body part would render any alleged "transitional" severely handicapped. A critter saddled with an appendage half-leg and half-wing couldn't fly or walk efficiently. An incrementally evolving eye would hardly contribute to fitness or survival. Dr. Michael Behe illustrated the problem by referencing a simple mousetrap with a single missing part---no mouse need fear, the trap couldn't function! Entropy The second law of thermodynamics doesn't track with Darwin's speculative "progress toward perfection." Given 4.55 billion years, the 2nd Law of Thermodynamics impacts the precise opposite of what evolution requires. Entropy's deterioration and decline is the inevitable consequence in nature. Homo sapiens age, grow weak and die; trees lose their leaves and die; cars rust; buildings crumble; mountains erode. Cataclysm Many extinct species disappeared abruptly, victims of violent, hydraulic cataclysm, rather than from the encroachment of newly-evolving descendants eliminating "inferior" ancestors as per Darwin's speculations. Numerous surviving species, still living today, escaped extinction, showing little modification from their fossil ancestors. |
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E interesant cum Mama Natura tine cu ursul, dar nu tine cu foca. Ma intreb ce camuflaj are o foca neagra pe gheata alba. Sau evolutionismul nu se aplica si la foci? |
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Daca prin absurd s-ar naste un urs verde - in genul gainii cu trei picioare - atunci anomalia nu s-ar transmite si generatiilor urmatoare. |
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Intrebarea mea era daca ursul alb aparut accidental a putut transmite genele mutante mai departe inainte de a patrunde in mijlocul intinderilor de zapada. |
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Din cate se stie cam toate mutatiile genetice actuale sunt anomalii. Ai cateva exemple de mutatii genetice pozitive? |
QUOTE (Figaro @ Sep 3 2003, 04:30 PM) |
Intrebarea mea era alta. Prin ce mister "vede" blana ursului culoarea alba a zapezii? |
QUOTE ("Mi_) |
Daca a avut loc înainte de fecundare, descendentii vor fi tot gaini cu trei picioare. |
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Can genetic mutations produce positive changes in living creatures? Are mutations responsible for evolution from amoeba to man? Evolutionists have ascribed wondrous powers to mutations, the ability to create new body parts and new animals (amoeba to man evolution). In reality, mutations are extremely dangerous and are wreaking havoc on the human race and other living creatures. It is held by evolutionists that genetic mutations are an avenue of positive change in living organisms. For example, Richard Dawkins' book, The Blind Watchmaker, seeks to establish a godless cosmos of chance in which the appearance of design in life has occurred by accident, by the incremental accumulation of positive changes in genes. His evidence relating to biochemical genetics, however, consists of theoretical models of little relevance to the real world. Thus, the question remains: What do we actually see in the world around us when we use scientific tools of measurement and observation? Do we see this "blind watchmaker" at work in any real-life examples, or do we see the opposite? The purpose of this article is to demonstrate the poverty of evolutionary theory to explain the facts in one well-researched area of biology--that is, the area of human genetics. It will show how the facts unearthed by this research show mutations to be, not a "blind watchmaker," but more truthfully analogous to a "blind gunman." The human mutation problem is bad and getting worse. Literally thousands of human diseases associated with genetic mutations have been catalogued in recent years, with more being described continually. A recent reference book of medical genetics listed some 4,500 different genetic diseases. Some of the inherited syndromes characterized clinically in the days before molecular genetic analysis (such as Marfan's syndrome) are now being shown to be heterogeneous; that is, associated with many different mutations. This review will only scratch the surface of the many recent discoveries. Still, the examples cited will illustrate a compelling general principle which extends throughout this expanding field. What are mutations? Mutations are defined as random changes in cellular DNA. They change the genetic code for amino acid sequence in proteins, thus introducing biochemical errors of varying degrees of severity. Mutations have been classified as deletions (loss of DNA bases), insertions (gain of DNA bases), and missense or nonsense (substitution of a DNA base). If the mutations affect germ cells (female ova and male spermatozoa), they will be passed to all the cells of the offspring, and affect future generations. Such mutations are called "germline mutations," and are the cause of inherited diseases. Mutations also occur in other populations of body cells and will accumulate throughout a lifetime without being passed to the offspring. These are called "somatic mutations," and are important in the genesis of cancers and other degenerative disease processes. What are the real world results of mutations? (examples) To survey the mutation problem, it will be helpful to consider a few examples of how mutations work their biochemical havoc. Cholesterol-related mutations In the cardiovascular system, it has long been recognized that a high circulating cholesterol content in the blood is associated with degeneration and narrowing of large and medium-sized arteries. this process is called "atherosclerosis" and is a leading cause of heart disease. More recently, a genetic biochemical defect causing hereditary high blood levels of cholesterol has been discovered and is know as "familial hypercholesterolemia" (FH). This disorder has been traced to mutation of a gene coding a transmembrane receptor protein. The gene is on chromosome 19 and has about 45,000 base pairs with 18 exons. Its encoded receptor protein is anchored in the membranes of all body cells, and allows them to capture and take in "packages" of fats and cholesterol (called "low-density" lipoproteins," or LDL) that are manufactured in the liver. The receptor protein has 772 amino acids which form five functional domains. At least 350 different disease-producing mutations of the cholesterol receptor have been described. These may be classified according to the affected functional domain. Examples of mutations in the cholesterol-related LDL receptor gene. Shown are the 18 exons of the gene and various combinations of known mutations, classes 1-5. (This illustration is adapted from a more detailed diagram in J.L. Goldstein and M.S. Brown, "Familial Hypercholesterolemia," in C.R. Scriver , A.L. Beaudet, W.S. Sly, and D. Valle, editors, The Metabolic Basis of Inherited Disease, 6th edition (New York: McGraw-Hill, 1989), p. 1232.) In the first class of mutation, little or no receptor is synthesized at all. In the second, receptor protein is synthesized, but does not take its proper place in the cell membrane. Third, receptor protein is present in the membrane, but does not link with the LDL packages. Fourth, the receptor protein is unable to stay in the membrane. Fifth, receptor protein is present in the membrane and links with the LDL packages, but does not bring them into the cell. None of these are beneficial. All body cells need cholesterol for their membranes, so a certain amount is necessary and good. However, defects of this receptor protein result in high blood levels of cholesterol through a feedback loop. When the receptor protein is not working, the cells keep on sending the signals for more cholesterol packages, and the liver complies. In homozygotes, cholesterol levels are three to five times the proper level, while heterozygotes have about twice the proper level. This results in rapid atherosclerosis, sometimes resulting in fatal heart disease in childhood. Cystic fibrosis mutation A second example is a common genetic disease, cystic fibrosis (CF). This multisystem disease cripples children and leads to early death. It damages the lungs, digestive organs and, in the male, the vas deferens (spermatic duct). Its differing effects, from mild to severe, are in part due to different types of mutation affecting one key gene. This biochemical basis is the mutation of a gene coding for a transmembrane protein regulating chloride ion transport across the cell membrane. This gene has 250,000 base pairs and is called the CFTR gene. It codes for a transmembrane protein of 1,480 amino acids. Research on this gene showed a mutation, delta-F508, occurring in most clinical cases of CF. This mutation is a deletion of three nucleotides resulting in loss of phenylalanine residue at position 508 on the peptide chain. Normal DNA . . . T ATC ATC TTT GGT GTT Cystic Fibrosis DNA . . . T ATC AT- --T GGT GTT In addition to this fairly common mutation, over 200 other mutations of this gene have been described. Just a few of these are associated with the more severe forms of the disease, which lead to early death from lung infections. Other mutations or combinations of mutations lead to lesser disease states, like chronic pancreatitis or male infertility, but again, no beneficial results have been observed. Cancer As a broad example of disease produced by acquired somatic mutations, let's consider cancer. The link between carcinogenesis and genetic mutation has become much clearer. Carcinogens (agents causing cancer) also tend to be powerful mutagens (agents producing mutations). The discovery of "oncogenes" and "tumor suppressor genes" has shown how this relationship works. Basically, these genes are concerned with regulation of the cell cycle. The oncogenes drive the process of cell replication forward, while the tumor suppressor genes hold it back. Both are necessary for proper cell function and growth. But mutational damage to components of both systems may produce an uncontrolled growth of cells, which is cancer. This phenomenon may be compared to a car in which there is damage to the gas pedal, causing it be be stuck "on," while the brakes are damaged at the same time. These mutations are usually acquired over decades, so cancer is mainly a disease of old age. However, studies have shown that inherited germline mutations of key oncogenes or tumor suppressor genes can predispose persons to development of cancers in childhood. Examples of this include childhood cancers like retinoblastoma, as well as familial cases of more common cancers (e.g., breast or colon) that have been linked to specific mutant genes (e.g., the BRCA1 and BRCA2 genes for familial breast cancer, and the APC gene for familial colonic polyps and cancers). Do mutations have any positive results? With this array of human diseases that are caused by mutations, what of positive effects? With thousands of examples of harmful mutations readily available, surely it should be possible to describe some positive mutations if macroevolution is true. These would be needed not only for evolution to greater complexity, but also to offset the downward pull of the many harmful mutations. But, when it comes to identifying positive mutations, evolutionary scientists are strangely silent. Sickle cell anemia The mutation responsible for sickle cell anemia has been put forward as an example of Evolution. The problems with this are obvious, as the sickle cell mutation, like the many other described hemoglobin mutations, clearly impairs the function of the otherwise marvelously well-designed hemoglobin molecule. It can in no way be regarded as an improvement in our species, even though its preservation is enhanced in malaria-endemic parts of central Africa by natural selection. Cancerous cellular degeneration Even more strangely, the process of cancerous cellular degeneration has been vied as a Darwinian form of mutation! Again, this idea fails to hold up under scrutiny. Malignant cells can hardly be considered to be an improvement over their normal counterparts. They are "fitter" only in their replicative activity, but even this is just an exaggerated use of already existing cellular machinery. In many other important ways, they have degenerative features. They show no gain of information, but generally a loss or disorder of functions. In all this research, not one mutation that increased the efficiency of a genetically coded human protein has been found. Conclusions What conclusions may be drawn from these few examples, and countless others like them? First, that the human mutation problem is bad and getting worse. Second, that it is unbalanced by any detectable positive mutations. To summarize, recent research has revealed literally tens of thousands of different mutations affecting the human genome, with a likelihood of many more yet to be characterized. These have been associated with thousands of diseases affecting every organ and tissue type in the body. The medical descriptions of many forms of inherited disease have a common theme: 80-90% of cases have affected individuals in the family tree, but the remaining cases are sporadic--the result of ever increasing numbers of new mutations. In all this research, not one mutation that increased the efficiency of a genetically coded human protein has been found. Mutations behave like a "blind gunman," a destroyer who shoots his deadly "bullets" randomly into beautifully designed models of living molecular machinery. Instead of a "blind watchmaker," the mutations behave like a "blind gunman," a destroyer who shoots deadly "bullets" randomly into beautifully designed models of living molecular machinery. Sometimes they kill. Thus, the "blind watchmaker" is an illusion. Worse than that, it is the intellectual and moral equivalent of an idol--an invention of the imagination, to which superhuman powers are falsely ascribed. Decay and degeneration This research affirms the reality of the past Biblical curse of decay and degeneration on the world of nature, as stated in both the Old and New Testaments. It also highlights the grim reality of the future hopelessness of the human race without the saving intervention of God and His Christ. Mutations continue to slowly harm us. Each generation has a slightly more disordered genetic constitution than the preceding one, and no amount of eugenics can reverse this process of decay. Gene therapy may mask the effects, but it will not reverse the underlying degenerative process. Mutations will eventually turn the human genetic code to gibberish. A slight but definite ongoing mutation rate, accompanied by a zero rate of positive genetic change, will eventually turn the human genetic code to gibberish. The problem is like a large book, written with perfect grammar in the beginning, but with random letter substitutions introduced at an ongoing rate. The book will still be readable for some time, but it will eventually lose all sense. Just as the universe is projected to reach a state of maximum entropy, so also the human race is condemned to a degenerative death, not just as individuals, but as a whole. In conclusion, the Christian hope stands as the only light in the darkness. Only the creative and regenerating work of Christ, as shown in His creation of all things (John 1:3), His miraculous healings, and in His resurrection from the dead, offers humankind true hope for the future. References Richard Dawkins, The Blind Watchmaker: Why the Evidence of Evolution Reveals a Universe Without Design (W.W. Norton and Co., 1987). Lubert Stryer, Biochemistry, 3rd edition (W.H. Freeman and Co., 1998). C. Koch and N. Hoiby, "The Pathogenesis of Cystic Fibrosis," The Lancet, Volume 341 (1993). Friedman Cohn, et al., "Idiopathic Chronic Pancreatitis and Mutations of the Cystic Fibrosis Gene," The New England Journal of Medicine, Volume 339 (1998). Robert Weinberg, "Oncogenes and Tumor Suppressor Genes," CA: A Cancer Journal for Clinicians, Volume 44, Number 3 (1994). Felix Mitelman, "Chromosomes, Genes, and Cancer," CA: A Cancer Journal for Clinicians, Volume 44, No. 3 (1994). J. Defasche and J. Kastelein, "Molecular Epidemiology of Familial Hypercholesterolemia," The Lancet, Volume 352 (1998). Robbins, Cotran and Kumar, The Pathologic Basis of Disease, 5th edition (Philadephia: W.R. Saunders Co., 1994). [ If this information has been helpful, please prayerfully consider a donation to help pay the expenses for making this faith-building service available to you and your family! Donations are tax-deductible. ] |
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KANGAROOS - Where do they come from? Why do they hop? Did they evolve from some other animal? Kangaroos are the symbol of Australia. They adorn its postage stamps, coat-of-arms, coinage, and even its major international airline. At the zoo or in their natural habitat of Australia (and New Guinea), they remain the most recognized and obvious of Australia’s fauna. Their faces, the way they carry their young in a pouch, their phenomenal leaping power, and their deadly ‘karate kicking’ have long intrigued people. The whole family is best known as the Macropodidae—literally the ‘big-footed’ family. This includes not just the six largest living species commonly called ‘kangaroos’, but also a further 48 species found in Australia alone, and another 13 found in New Guinea—67 modern species in all. The range of two Australian species, the agile wallaby and the red-legged pademelon, spills into New Guinea as well. The term ‘modern’ is applied because this vast empire was once much greater, with over 100 species in Australia alone.1 The term ‘wallaby’ is applied to those species where the adult male has a body mass less than about 20 kg (44 lbs) and feet less than 25 centimeters (10 inches) long. However, scientists can recognize no major anatomical difference between these and kangaroos. Kangaroos’ superb design, their sophisticated reproductive methods and their amazing, energy-efficient locomotion did not come by any evolutionary process. For example, unless the pouch and the joey’s ability to find it were fully functional, they would have left no offspring. They varied enormously in size. The tiny, scampering musky rat-kangaroo still lives in the tropical rain-forests of northern Queensland (Australia). However, the massive, blunt-faced Procoptodon is extinct. Three basic size ranges are recognized today. At the other end of the scale from the six large types mentioned above are the rat/rabbit-sized bettongs, potoroos and rat-kangaroos. In between are the tree kangaroos (a specialized group comprising nine species that live and move about in the trees), and those commonly called wallabies. Kangaroo Reproduction Why the pouch? In the desert species, carrying the baby in the pouch is convenient for the female, who may travel many miles for fresh food and water. The youngster stands a greater chance of survival because it does not have to keep up with her and is tucked away from predators. During prolonged drought, kangaroos stop breeding. In some species, a doe [the female] is able to delay the development of a fertilized egg inside her until an older joey dies or vacates the pouch. This remarkable phenomenon occurs in the red kangaroo, the eastern grey kangaroo, the common wallaroo (euro), the brush-tailed bettong, and several of the larger wallabies. It has also been noted in the honey possum and some non-marsupial mammals such as bats and seals.2 Another incredible aspect is that the doe can determine the sex of her offspring. How she does this is unknown, but she tends to put off bearing males until she is older. Males move away after about two years, but females stay with their mothers longer and benefit from ongoing support.3 A doe is nearly always pregnant. From sexual maturity to death, she is rarely without three offspring — an embryo in the womb, a joey in her pouch, and a larger youngster at her heels. The joey is born after a gestation period of about 35 days (depending on the species) and in the largest species is the size of a human thumb nail. In the smallest, it is only the size of a rice grain. Naked, blind and deaf, it must make its way unaided from the birth canal to the pouch. All going well, the climb will take less than 10 minutes. The joey can survive only a few minutes unless it reaches the pouch and attaches to one of the four nipples. Once there, its mouth swells on the nipple so that it cannot be removed without injury. A ring of strong muscles, similar to human lips, seals off the opening to the pouch to protect the joey from bouncing out, and keeps the pouch waterproof if mother goes for a swim.4 After three months, the developed joey emerges from the pouch to make short trips in the outside world. However, it will return to the pouch to suckle and sleep until eight months old. How fast are kangaroos? Why do kangaroos hop? Over the years, scientists have put forward theories concerning the hows and whys of kangaroo locomotion. As yet, none has fully explained every aspect. Hopping appears to be more energy-efficient than running or galloping. The faster kangaroos hop, the less energy they use for the same distance. Treadmill studies have shown that kangaroos maintain a constant number of hops per minute. Regardless of how much the treadmill speeds up, they simply take longer and longer hops. Kangaroos function much like bouncing balls. A ball will bounce a number of times without a fresh input of energy. Every time it hits the ground, some of the energy is shifted to the rubber, stored there, then recycled in an elastic bounce. Jumping kangaroos store 70% of their energy in their tendons, compared to running humans, who can store and reuse only about 20%.4 A hopping kangaroo also uses less energy to breathe than one standing still. Part of the secret lies in the way the abdominal organs ‘flop’ within the kangaroo’s body. Instead of using muscle power, air is pushed out of the lungs by the impact of the organs against the diaphragm at each landing. Efficient travel is very beneficial to arid-dwellers such as the Red and Western grey kangaroos, the Tammar wallaby and the euro, which may need to travel long distances between water and feed. However, many species inhabit timbered country, with abundant food and regular rainfall. Evolved from possums? The Macropod family is alleged to have evolved from either the Phalangeridae (possums) or Burramyidae (pygmy-possums) during the so-called Oligocene epoch some 30 million years ago.5 However, there are no fossils of animals which appear to be intermediate between possums and kangaroos. Wabularoo naughtoni, supposed ancestor of all the macropods, was clearly a kangaroo (it greatly resembles the potoroos which dwell in Victoria’s forests).6 If modern kangaroos really did come from it, all this shows is the same as we see happening today, namely that kangaroos come from kangaroos, ‘after their kind’. A stunning example of this is the modern rock-wallabies. When John Gould first made notes of these animals last century he mentioned only six species. Later ten species were counted, and now a total of 15 are recognized. Current research is indicating that these wallabies are still splitting into new species.7 However, such instances of one group ‘splitting’ into more groups is not evolution, as Creation magazine has pointed out repeatedly. The reason is that no new genetic information arises during such events. Creationists have postulated that such speciation must have happened many times after the Flood, as populations of creatures separated by valleys or mountain ranges have adapted to environmental conditions within their territories. Some of the original population’s genes enable their owners to survive in their particular environments, while other genes are lost to such natural selection. However, all the genes were present in the original population. Each ‘daughter’ population carries somewhat less of this information, so is less able to respond to future environmental changes.8 They are all still rock-wallabies, and these changes did not take millions of years. In fact, seeing such ‘adaptive radiation’ happen so quickly is a great boon to creationist models. It shows how there would have been ample time since Noah’s Flood for all known kangaroo types to have come from one or a very few original kinds.9 Evolutionists explain the wide variety of kangaroos and their specialised survival methods as millions of years of trial and effort, chance mutation and selection. However, kangaroos’ superb design, their sophisticated reproductive methods and their amazing, energy-efficient locomotion did not come by any evolutionary process. For example, unless the pouch and the joey’s ability to find it were fully functional, they would have left no offspring. Australia’s extinct giants Australia once had many marsupials much larger than those remaining today. The ‘giant wombat’ Diprotodon is probably the best-known of these. The giant kangaroo Procoptodon could stand three metres (ten feet) tall. They (and also a non-marsupial, the bird Genyornis, a larger version of the emu) are collectively called Australia’s extinct ‘megafauna.’ What happened to all of these? Many have ‘devolved’ down to smaller representatives. For instance, today’s red kangaroos and Tasmanian devils are much smaller than their fossil counterparts. A recent find at Cuddie Springs in New South Wales, of human tools together with the bones of some of these megafauna, raises the suspicion that people helped drive them to extinction, which of course is no surprise for creationists. Tests have confirmed that some blood is still present on the tools, which suggests that it was probably nowhere near as long ago as evolutionists say. |
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THE HORSE In 1841, the earliest so-called "horse" fossil was discovered in clay around London. The scientist who unearthed it, Richard Owen, found a complete skull that looked like a fox's head with multiple back-teeth as in hoofed animals. He called it Hyracotherium. He saw no connection between it and the modern-day horse. In 1874, another scientist, Kovalevsky, attempted to establish a link between this small fox-like creature, which he thought was 70 million years old, and the modern horse. In 1879, an American fossil expert, O. C. Marsh, and famous evolutionist Thomas Huxley, collaborated for a public lecture which Huxley gave in New York. Marsh produced a schematic diagram which attempted to show the so-called development of the front and back feet, the legs, and the teeth of the various stages of the horse. He published his evolutionary diagram in the American Journal of Science in 1879, and it found its way into many other publications and textbooks. The scheme hasn't changed. It shows a beautiful gradational sequence in "the evolution" of the horse, unbroken by any abrupt changes. This is what we see in school textbooks. The question is: "Is the scheme proposed by Huxley and Marsh true?" The simple answer is "No". While it is a clever arrangement of the fossils on an evolutionary assumption, even leading evolutionists such as George Gaylord Simpson backed away from it. He said it was misleading. So what's the difficulty for the horse with the theory of evolution? If it were true, you would expect to find the earliest horse fossils in the lowest rock strata. But you don't. In fact, bones of the supposed "earliest" horses have been found at or near the surface. Sometimes they are found right next to modern horse fossils! O.C. Marsh commented on living horses with multiple toes, and said there were cases in the American Southwest where "both fore and hind feet may each have two extra digits fairly developed, and all of nearly equal size, thus corresponding to the feet of the extinct Protohippus". In National Geographic (January 1981, p. 74), there is a picture of the foot of a so-called early horse, Pliohippus, and one of the modern Equus that were found at the same volcanic site in Nebraska. The writer says: "Dozens of hoofed species lived on the American plains." Doesn't this suggest two different species, rather than the evolutionary progression of one? There is no one site in the world where the evolutionary succession of the horse can be seen. Rather, the fossil fragments have been gathered from several continents on the assumption of evolutionary progress, and then used to support the assumption. This is circular reasoning, and does not qualify as objective science. The theory of horse evolution has very serious genetic problems to overcome. How do we explain the variations in the numbers of ribs and lumbar vertebrae within the imagined evolutionary progression? For example, the number of ribs in the supposedly "intermediate" stages of the horse varies from 15 to 19 and then finally settles at 18. The number of lumbar vertebrae also allegedly swings from six to eight and then returns to six again. Finally, when evolutionists assume that the horse has grown progressively in size over millions of years, what they forget is that modern horses vary enormously in size. The largest horse today is the Clydesdale; the smallest is the Fallabella, which stands at 17 inches (43 centimeters) tall. Both are members of the same species, and neither has evolved from the other. My research has left me troubled. Why do science textbooks continue to use the horse as a prime example of evolution, when the whole schema is demonstrably false? Why do they continue to teach our kids something that is not scientific? Dr. Niles Eldredge, curator of the American Museum of Natural History, has said: "I admit that an awful lot of that (imaginary stories) has gotten into the textbooks as though it were true. For instance, the most famous example still on exhibit downstairs (in the American Museum) is the exhibit on horse evolution prepared perhaps 50 years ago. That has been presented as literal truth in textbook after textbook. Now I think that that is lamentable ...". I agree. -------------------------------------------------------------------------------- The horse series is often presented as proof of evolution. The number of toes in foreleg and hind leg supposedly decreased as the horse evolved, and the size supposedly increased from a small doglike horse to a large modern horse. Yet three-toed horses have been found with one-toed horses, showing they lived at the same time. And there are tiny living Fallabella horses only 17 inches ( 43 centimeters) tall. |
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Evolutionists explain the wide variety of kangaroos and their specialised survival methods as millions of years of trial and effort, chance mutation and selection. However, kangaroos’ superb design, their sophisticated reproductive methods and their amazing, energy-efficient locomotion did not come by any evolutionary process. |
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For example, unless the pouch and the joey’s ability to find it were fully functional, they would have left no offspring. |
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In the desert species, carrying the baby in the pouch is convenient for the female, who may travel many miles for fresh food and water. The youngster stands a greater chance of survival because it does not have to keep up with her and is tucked away from predators |
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Figaro: Mi_, se pare ca tu confunzi o specie cu o rasa. Ursul alb este o variatiei a speciei in cazua, dupa cum acelasi lucru se intampla si cu rasele umane, dar a sustine ca o broasca se transforma in cal pe parcusul a milioane de ani mi se pare absurd. |
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Volume 8, No 1, Spring 1998 METHODOLOGICAL PROBLEMS OF EVOLUTIONISM Ümit Dericioglu Introduction It is important to make a sound logical assessment of the methods used in advancing a theory. The axioms of a theory need not be self-evident. However, they should not be mutually contradictory. A good theory should explain the unexplained before, in a way which is not arbitrary and should predict unknown phenomena. The process of deduction leading to explanations and predictions should be done with sound logic devoid of contradictions and fallacies. It should also be remembered that theories or new ideas proposed by scientists are not independent of the dominant world views or philosophies of their time. The Underlying Philosophy Evolutionary ideas did not start with Darwin. There were attempts before to explain how the living world came to be through some evolutionary processes. Lamarckism, the theory of inheritance of acquired characters was one such attempt. What was common among the evolutionary ideas including Darwinism, was the naturalist belief which still prevails in science today. Scientists believe that every natural event has natural cause(s). Although this belief itself can never be proven true through experimental sciences, it is believed that scientific research would not be possible if we stopped asking for an explanation, by attributing it to a supernatural or metaphysical cause. In addition to the fact that an infinite regression of cause and effect is not logically possible, this naturalist presupposition is not even necessary for scientific research. Ironically, evolutionism based on naturalism can even hinder the research possibilities, as this will be shown below. What was different in Darwinism, however, from the other evolutionary ideas was the concept of "survival of the fittest" which had begun as "survival of the strongest" and ended up as "natural selection". Interestingly almost all of men who propounded some idea of natural selection, in the first half of 19th century were British. Darwin's and Wallace's views reflected the widespread belief of their time in their country, in the progress through competition.[1] Russian zoologist, Pyotr Kropotkin [2] who was a staunch Darwinist volunteered to serve in the military in Siberia, in order to observe the struggle for the resources among the animals for survival. What he found instead was harmony and cooperation which later he depicted in his famous book Mutual Aid. It was obvious that Darwinian picture of nature did not fit the reality. Natural Selection In Neo-Darwinism, the central feature of selection is the differential reproduction. Natural selection occurs at population level, not at individual level. A part of population with advantageous trait(s) which manage to survive under otherwise difficult conditions pass their genes onto their progeny. They thus render their offspring dominant in the populations of future generations. Since the early days of Darwinism, a very serious objection has persisted: Natural selection is a tautologous concept. In other words, it means those which can survive survive and pass their advantageous traits which enabled them to survive to their offspring. The tautology is obvious here. The concept of natural selection does not say anything new. The most important pillar of Darwinism is logically defective! In response to this criticism, evolutionists claim that the question is not whether natural selection is a tautology but whether it is the guiding force of evolution. In other words, does the evolution occur in a particular environment as a result of natural selection? According to Hull, a lack of reference to environment deprives the theory of its empirical content.[3] Yet the tautology is still there. Besides, the natural selection is still hardly empirical. How shall we know what advantageous or deleterious mutations an organism might develop? With what probability? Since we need to take the relationship with environment into account, how do we decide whether a certain environmental condition is fatal for one part of a population while it is not for another? From the moment we know that there is a trait for an otherwise fatal condition, the tautology is there again. Before the trait and condition come about we cannot predict them. Once they occur, there is nothing new to know. Some evolutionists regarded the natural selection as the sole cause of evolution while some completely rejected it likening to the ill-fated phlogiston.[4] In fact, it is even worse a concept than phlogiston. Phlogiston had been eventually discredited in experiments. Natural selection, however, due to its tautological nature, can never be proven wrong. Indeed, natural selection provides explanations or plausible speculations for everything like phlogiston did. It is a panacea to explain the order and purpose in organisms without resorting to teleology. However, it is a concept which did not contribute to science. On the contrary, with its phlogiston-like all-encompassing explanations, it has been the weakest point of Darwinism. Darwinism (or Neo-Darwinism) with a logically defective and unscientific concept such as natural selection as an important pillar has been hardly a theory. Perhaps it was a poor attempt which was hoped to pave way to a successful theory of origins some day. Avoiding The Real Issue Evolutionists always considered the mutations capable of providing endless combinations, a viable few of which to be selected generation after generation, thus resulting in substantial changes and novelties in organisms. However, the real scientific issue was the nature of the mutations. Were they really random? Were there limits to the changes by mutations? In other words, were the changes a possible manifestation of limited genetic potential? Actually, nobody knows if the genes can lead to endless possibilities. This was just an assumption by evolutionists. According to them, when combined with very long time and filter of natural selection genes are capable of generating many highly ordered and sophisticated systems. A cautious reader will see the fallacious reasoning here: an endless random variant generator such as genes plus a sieve as natural selection, given enough time is capable of generating any existing system. By using the same fallacious logic one can explain anything one wants. For example, the origin of life before the genes were formed can also be explained. However, this is not science, but only an assumption of evolutionist.[5] The fallacious reasoning of evolutionists is a good example of how naturalist philosophy may lead to unscientific claims contrary to the widespread belief that science is only possible with naturalist presuppositions. The Definition of Species The lack of a spatio-temporally independent definition of species presents a problem in evolutionism. For example, mass is the resisting capability of matter to force in Newtonian physics. The definition is always the same, regardless of time and space in question. Since the publication of The Origin of Species the evolutionists have been speaking of the evolution of species without being able to give a clear, unproblematic definition of "species". A very popular and widely accepted definition of species is that organisms that can interbreed are of the same species. An interesting result of this definition is no matter how similar the creatures are, they are not of the same species if they cannot interbreed. Actually this definition is related to Mayr's Founder Principle. According to this idea, if a small subset of a population is isolated from the main population for some reason, since they would represent only a small subset of the gene pool, they would start to diverge and become sexually incompatible with the main population. Indeed, there are populations which do not interbreed after a long isolation, or if they are forced to, their offsprings are either sterile or genetically defective. It is said that the isolated population is on the verge of speciation. In fact, according to the definition above, they are almost new species, even if they look the same, behave the same. However, the similarity between some creatures such as squirrels and moles, etc. in Australia and those others in other continents is at odds with Founder Principle. Evolutionists would like to explain it away by calling it "parallel evolution" which results in similar creatures under supposedly similar conditions. Unfortunately, parallel evolution contradicts Founder Principle. Aborigines who are believed to come to Australia, 30-40 thousand years ago can interbreed with Westerners. Therefore they are of the same species according to the definition. Why did such a long isolation not cause a genetic incompatibility? Unless Mayr's Founder Principle explains these problems and makes specific claims as to which species under what conditions become genetically incompatible after how long isolation in a consistent and testable way, it is merely an interpretation of a fact with the hope of explaining a process of so-called speciation. Another problem the above definition represents is its inability to cover asexual species of our time and extinct species of the past. In case of asexual species there are no male and female individuals of the same species. Therefore the definition of interbreeding capability does not work for them. As for the extinct species of the past, how do we know if they were different from those which are similar today? Take Neanderthals, for example. Could they have interbred with modern humans? If they could, they would only be a different human race! How, then, can we conclude that some hominids represented the missing link of a different species on the way to homo sapiens, if we are to stick to the above definition? Form/Function - Hardware/Software In order to make a healthy comparison between the species we need to take not only the form into account but also the function. Unfortunately the fossil record gives us snapshots of a subset of past organisms. But it does not tell us much about the internal structure and function of the organs and behaviour of those creatures. Even a simple-looking change such as extending the neck of giraffe requires extensive adaptations in the body. Not only would the blood vessels extend and adapt accordingly, but the heart should also evolve strong enough to pump the blood to the brain. Unless we have a holistic approach in studying the organism, we would only see a perhaps deceptively small part of the whole picture. Therefore it seems logically absurd to make broad statements about the history of the biological world, based on the fossil record which is a small subset of past flora and fauna. Besides, that small subset is only a part of form devoid of function. A good example of this is the Coelacanth. It was believed to be extinct for millions of years until it turned up in the nets off the coast of Madagaskar, in 1938. Because it is a lob-finned fish, evolutionists claimed (and still do) that amphibians evolved from it. In 1986, a German biologist, Hans Fricke, studied the behaviour of the fish, in deep sea, using a specially designed submarine. He found that the fins enabled the Coelacanth to swim in all directions. They had nothing to do with the way the amphibians crawled.[6] With the advent of computers and neurology, it is now known that the biological systems have a "software" part which governs their organs and behaviour. For example, even if a person has a complete mouth, tongue and vocal cords, i.e. "the perfect hardware", he cannot speak if the speech center in the brain is defective. Similarly, flying and navigating capabilities of birds would not be possible, unless their brains were equipped with the appropriate "software". Evolution of the "software" in harmony with the evolution of "hardware" through random blind coincidents of natural events is impossible to explain because the probability of such a parallel development of "hardware" and "software" through random events is practically zero. Therefore, "software" part of organisms presents an insurmountable challenge to the evolutionists. More Fallacies: Argument From Similarity, Argument From Sequence In order to establish an ancestral relationship between two species, evolutionists look for the similarities. Logically, however, if similarity indicates relationship, then dissimilarity should indicate the otherwise.[7] Evolutionists, while freely using the similarities to claim the evolutionary relationships, ignore the dissimilarities. In other words, their logic works only for their theory, never against it! A striking example is hemoglobin. Red cells of humans have an antigen which is indistinguishable from those of apes. But when we find the hemoglobin also in root nodules of leguminous plants, the fallacy becomes obvious.[8] Supposing that the fossil record indicates stages from primitive (multi-cellullar) creatures to more complicated ones in time, evolutionists claim that evolution is a fact. Even the ones who admit there are serious problems with Darwinism, claim that explaining the process of evolution, i.e. Darwinism is one thing and the fact that evolution occurred is another. According to them, the fossil record undeniably points to that fact.[9] However, this line of reasoning shows how assertions laden with belief or philosophy can be portrayed as facts. Despite the fact that the creatures can be sequenced from primitive to more sophisticated in time, it does not necessarily follow that they evolved from each other. Take the example of an imaginery human tribe which produce pots and pans. As their skill and technology advance they develop more sophisticated ones. Later, for some reason, the tribe becomes extinct and in time, their pots and pans become fossilized. Then, very evolutionist minded aliens land and dig up the fossils. Their advanced methods date the simpler ones before the sophisticated ones. Then, aliens safely conclude that the evolution of pots and pans is a fact! The only fact the fossil record tells us is the existence of some creatures in geological ages, provided that the relative datings are accurate enough. How they came into existence, whether they were re-designed from the preceding creatures or evolved from them or some of them showed up suddenly is a completely different matter. The Lack of Scientific Method Darwinism or Neo-Darwinism is claimed to explain the process. Unfortunately, however, there is no unambiguous method expressed in logical or mathematical language. All of the explanations are arbitrary in nature. If the process was really known as they always claim, we would be able to make clear scientific deductions. Then, it would be possible to feed the characteristics of a species and and some well defined conditions as an input to the process and learn the output. That would not have to be an imaginary species to be evolved millions of years later. But, at least, it would give us the tree of relationship of existing species and of the fossils. Then, it would be possible to even predict some yet-undiscovered species. However, Darwinism is nothing of that sort of theory. On the contrary, evolutionists prepare their taxonomical trees with evolutionary glasses and then tell us who evolved from whom! Delayed Research Possibilities Since evolutionists have always believed that mutations are open-ended possibilities, it never occurred to them that the genetic potential of a creature could be of a limited number of possibilities. Therefore, research to predict and test those possibilities could not be carried out. If we had a theory telling us the scope and the kinds of changes an organism could go through, it would be very fruitful. That way the breeders' practical knowledge would be generalized and enriched under the theory for all or many more species. Another delayed research topic was the so-called vestigial organs. Evolutionists regarded them useless remnants from the evolutionary ancestors until later they were found to serve important functions in the body. Now no cautious scientist can claim that an organ is useless. Only it can be said that its purpose is not known yet. The two cases above are simple but good examples to show how Darwinism based on naturalism can block or delay some avenues of scientific research. Conclusion As seen above, evolutionism suffers from many logical and methodological problems. It has a goal of explaining the origins and development of the living world. Yet the component parts of it are weak speculations which are often inconsistent and logically defective. Naturalism and the urge to find answers for the origins and the positivist belief in science that it would provide the answers on all natural phenomena sooner or later have led scientists to areas beyond their capability. Evolutionists attempted the Herculean task of explaining the impossible, the occurrence of immensely organized hardware and very sophisticated software, the information of which was uniquely hard-coded during each regeneration. And they have ended up with speculative, self-contradicting and fallacious claims. Evolutionism has reversed their view from a teleological paradigm to a self-ordering paradigm of natural selection. This tautological paradigm has spread to other research areas to provide explanations through almost arbitrary speculations. Evolutionism, under the disguise of being purely scientific, has abused science in order to advance its underlying philosophy. Long ago, a man of wisdom and a reputable scientist, J. W. Dawson had warned evolutionists of this ill-fated path they have been driving science through: "Nothing can be more interesting in a psychological point of view than to watch the manner in which some of the strongest and most subtle minds of our time exhaust their energies in the attempt to solve impenetrable mysteries, to force or pick the lack of natural secrets to which science has furnished no key." [10] and, "It is a great mistake here to suppose that a little knowledge is dangerous; every grain of pure truth is precious and will bear precious fruit. The danger lies in misusing the little knowledge for purposes which it cannot serve." [11] We will perhaps never scientifically know the history of life on Earth, how it began and how it developed. Claiming that evolution is the only alternative to Biblical special creationism which is not acceptable and unscientific, is yet another fallacy. Norman Macbeth, in his Darwin Retried, calls it "best-in-field fallacy". He says: "Is there any glory in outrunning a cripple in a foot race? Being best-in-field means nothing if the field is made up of fumblers." [12] Marxism claimed to explain human history through a dialectic materialist economic paradigm. The paradigm was nothing but a reaction based on a materialistic philosophy against the Western Capitalism and Colonialism. The Western Capitalism and Colonialism, in turn, began in a small part of the world and in a relatively very short time span of a long human history. Then Marx and his followers extrapolated their reactionary paradigm to all of the world and all times, naming it "scientific socialism". It was an over-simplification of human history as seen through their ideological glasses. Similarly, Darwinism made big claims about the long history and great diversity of life which is a very complex reality, by extrapolating the mentality of 19th century colonialist England to living nature and its origins. One should always receive with great caution big claims under the banner of science, modelled after some popular paradigm or philosophy of a particular era in the Western part of the world. |
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Evolutionists always considered the mutations capable of providing endless combinations, a viable few of which to be selected generation after generation, thus resulting in substantial changes and novelties in organisms. However, the real scientific issue was the nature of the mutations. Were they really random? Were there limits to the changes by mutations? In other words, were the changes a possible manifestation of limited genetic potential? |
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Evolutionismul propune mutatii aleatoare, care se selecteaza in functie de conditiile de mediu. |
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Un soft poate face un miliard de combinatii aleatoare, insa NICIODATA nu se va transforma de unul singur intr-un alt program. |
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Ideea unor greseli de compilare care pot "naste" linii de cod valabile e absurda. |
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Dar de aici si pana a sustine ca peste milioane de ani ursul brun va avea aripi de libelula, picioare de broasca si branhii e total deplasat. |
QUOTE ("Mi_") |
Nu evolutionismul propune mutatiile ci imperfectiunea mecanismelor de duplicare ADN în timpul diviziunii celulare. |
QUOTE ("Mi_") |
Soft-ul numit genom are proprietatea de a se multiplica singur. Deci de unul singur. |
QUOTE ("Mi_") |
Si datorita mutatiilor, poate sa se transforme în alt program (alt genom). Nu-i mare filozofie aici. |
QUOTE (Mi_ @ Sep 4 2003, 06:14 PM) |
La fel nu ai putea admite logic ca un nor încins de Hidrogen se poate transforma în frumusetea de monitor de pe biroul tau. |
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Se pare ca cineva a trebuit sa umble. |
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Si mai exista calcule si calcule, care arata ca aparitia vietii e mai mult decat o intamplare. E o minune. |
QUOTE ("Mi_") |
Uite ca eu pot admite ca un Word, lasat un milion de ani, cu o rata de duplicare de trei ori pe secunda si o rata de erori la duplicare de 10 la puterea minus 10, poate deveni Autocad, dar nu numai atât, poate deveni ceva la care nici nu te-ai gândit vreodata ca ar putea exista. Poate. |
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Repet intrebarea: da-mi un exemplu de mutatie genetica benefica, care sa nu genereze o maladie |
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Din nou refrenul evolutionist preferat: poate. Stii bine ca un asemenea lucru nu e posibil |
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Nu exista erori functionale, dupa cum nu exista nici zapada fierbinte sau gheata clocotita. |
QUOTE ("Catalin") |
Ma duc in Norvegia si fac niste copii pe-acolo. Unii dintre sufera o mutatie genetica si se nasc blonzi. |
QUOTE ("Catalin") |
Este foarte posibil. Iar data viitoare cind spui ca un lucru nu e posibil sa vii si cu ceva argumente. |
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Mutatie genetica e atunci cand te nasti fara maini sau picioare. E vorba de un accident. La asta m-am referit. Copii blonzi se nasc prin recombinari genetice, nu prin mutatii!!!! Un blond nu e un mutant. E vorba de diversitate in limite stricte, nu de accidente care genereaza specii noi!!! |
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Argumente? Ce argumente are evolutionismul? |
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Din punct de vedere al softului oricine stie ca un bug nu e un avantaj, ci un dezavantaj |
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O eroarea pozitiva nu e eroare. Exista diversitate - mi se pare normal, dupa cum exista si liber arbitru - dar un program genetic, la fel ca si un soft, nu-si va depasi propriile limite |
QUOTE ("Catalin") |
Mutatie inseamna orice modificare de ADN. |
QUOTE ("Catalin") |
Dupa cum ti-am mai zis, asta e valabil in cadrul programarii clasice in care programatorul stie exact la ce sa se astepte din partea soft-ului si nu apreciaza erorile. Dar, atunci cind programatorul nu stie exact unde trebuie sa ajunga, bug-urile pot fi avantajoase. |
QUOTE ("Catalin") |
Soft-ul nu are limite. |
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Nu exista insa copii cu parul verde si nici nu vor exista niciodata |
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toate combinatiile care se fac au loc doar in limitele designului initial |
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Pot fi avantajoase in sensul ca ii pot da idei - in cel mai bun caz - nu si ca programul se poate rescrie prin erori |
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Programatorul nu are limite. Softul are limite. |
QUOTE ("Catalin") |
Pe ce te bazezi? pe Biblie? |
QUOTE ("Catalin") |
Ba tocmai ca se poate! |
QUOTE ("Catalin") |
Regula de bun simt: Asa cum eu nu vin sa-ti vorbesc tie despre euharistie, nu-mi vorbi nici tu mie despre ce poate sa faca un program si ce nu poate! |
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Figaro: E ca in gluma cu tipul care a scapat de amenintarea cancerului. L-a calcat masina. |
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mutatia reprezinta un accident, o aberatie. |
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Wordul NU poate sa remixeze fisiere wav, nu poate sa compuna muzica, nu poate sa faca separatii pentru tipar. O va face doar daca un programator il va regandi. Insa ideea ca Wordul s-ar putea rescrie singur in milioane de ani e absurda. |
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Un design, care e gandit sa elimine erorile aparute. Mitul evolutionist al accidentelor benfice nu poate avea o justificare in prezenta unor limite precise. |